Breast Growth For Genetic Males

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Sorry to hear about your struggle, HoF.

Sounds like you are predisposed towards being very lean, have you considered volufiline cream for the breasts and hips? Over time it increases the amount of fat cells you have in the area you apply it.

For what it's worth I think long legs and a lean body is very attractive.

Hi again,

If you haven't been seen by a health care provider specializing in transgender care I'd recommend seeing one...and completing necessary lab work to determine baseline levels. If you've already seen a health care provider and gotten nowhere with them, find someone else. Honestly, imho the WPATH standard is archaic...I did the standard WPATH care (E2 + spiro + finasteride) and it made me feel miserable.

Spiro should initially be given for up to 6 month, it comes with many side effects and it also has poor breast outcomes. As a matter of fact the incidence of MTF seeking breast augmentation from using spiro is around 60-70%. Finasteride is a neurosteroid and can cause depression. There's something called finasteride syndrome that once users (of fin) go off the medication it takes months to feel normal.

My experience of trans care has been shoddy at best. I've faced just about every roadblock one can face for gender care, even at present I'm struggling to get the best care. I see a new health care team next week to monitor my transition. I've done the DIY route, basically got fed up dealing with gatekeeping doctors. It's hard finding good trans care for some people...then again some get lucky and find a stellar doctor who isn't afraid to step out the standard orthodoxy of archaic health care.

For me, my goal of getting the appropriate HRT meds should be as follows:

IM (intramuscular injection) of E2 (estradiol)
Micronized progesterone
Low dose Transdermal E2
No anti-androgens needed (Orchiectomy in August of 21)

The dosages will be determined between my doctor and myself based on lab work. After 7 years of being on HRT I know what amounts get me to the therapeutic level I want to achieve.

HandofFate, I would encourage you to review this study below. And the reason I listed this study is from the fact everyone metabolizes differently. As an ectomorph your needs might be unique as to others, meaning you might need more or less than the typical HRT meds. However, lab work can only determine those needs. Doing the standard lab tests of Testosterone + Estradiol + SHBG doesn't identify all the underlying conditions of why hormones don't reach minimum standard reference ranges. I will list the labs needed if so desired. My advice is simple, don't let the lack of progress defeat you, you've put all this time into pursuing a certain goal, why throw in the towel now?.

Achieving Physiologic 17-ẞ-Estradiol Levels in Transgender Females on Estradiol Transdermal Patches and Optimal DosingAchieving Physiologic 17-ẞ-Estradiol Levels in Transgender Females on Estradiol Transdermal Patches and Optimal Dosing
https://academic.oup.com/jes/article/5/S...87/6241193

Thank you Nipply and Phanatic for your advice.

Yes I have used several creams with volufiline and Zhi mu extract with noogleberry.

I don't want to be attractive. I need everyone on the planet to think I'm born female thanks.

I have tried everything but patches, I had to self med the transdermal progesterone and estradiol.

My doctor usually does everything I ask of him. The problem is you get D cups right away and I get NOTHING.

So why would you complain? My blood tests have always been good, except when I did oral estradiol alone, without being addition to injectable estradiol.

I am currently taking 10mg ev/week, 50mg bicalutamide, and 200mg rectal progesterone cycled.

With MSM powder, vitamind D3+K2,boron,low dose aspirin,Omega 3-6-9, have cycled red reishi.

I didnt want to go on this thread as I'm embarrassed.
When I went on pm and other stuff it took a while but I got to a very heavy and bouncy C cup verging on a D cup....fantastic! But I was a bit fat.
I now am very dangerously underweight as I have developed an eating disorder and I only just fill my B cup bras, it's all gone, all of it. We can dream of being huge and bouncy but honestly why not just be petite with a nicely formed body. To grow a pair I think needs fat intake......I'm probably way off now as I do everything wrong anyway it seems.

(27-11-2021, 10:08 AM)Drew Wrote: [ -> ]I didnt want to go on this thread as I'm embarrassed.

I don't think anything we can say is going to help this person. I feel bad for them. They're frustrated, and they wanted to come here to vent. I think implants are the best solution (yes, I know, 'I can't afford them', etc. etc.).

Implants look fake on male chests. I can always spot them. They look disgusting. I need natural breasts that sag down and cover my man chest so people think I'm born female.

(27-11-2021, 09:01 AM)HandofFate Wrote: [ -> ]So why would you complain?

Our last conversation (via pm) was in march ('21), I haven't heard from you since...don't be rude. I don't mind you addressing your concerns here but it sure looks like you'd rather vent.

(27-11-2021, 09:01 AM)HandofFate Wrote: [ -> ]The problem is you get D cups right away and I get NOTHING.

Honestly I feel bad for you, but I'm not the enemy here. In the past 3 years you've been on HRT have you ever discussed your frustrations with your doctor?...or even in Dr. Powers sub reddit? If you've had a conversation with your doctor I'd be interested in what his recommendations were. I do think it's worthwhile to see a therapist while on HRT, and usually most HRT healthcare providers insist on this course when starting HR, and continued counseling throughout one's HRT journey, or as needed.

You should've seen something happening in the first 6 months of starting HRT, whether it was feminization and/or breast growth...and apparently nothing happened. My thoughts are that for every 6 months without ANY results one has to reevaluate the program. The labs I'd ask for are as follows:

Total T
Free T
Estradiol
Free E2
SHBG
DHT
LH
FSH
Prolactin
Progesterone
DHEA
Cortisol

Additionally CBC (Complete Blood Count)

If you would, please post any recent labs. Look up Oki's program, she's tall and developed nicely.

Anyone embarking on NBE/HRT should understand that the upkeep of breasts doesn't end with achieving one's goal, they'll need to be maintained for the rest of their life. Granted HandofFate, you just want the chance to realize that first step. Keep in mind, the male chest is wide to start off with, even D cup's won't hide that fact.

Thanks Drew and Nipply for offering advice.

My doctor has changed up my dosage and added real progesterone, and also increased that dose to 200mg.

I have seen a gender therapist they are extremely expensive and do nothing.

crazy how D cups don't look tiny on your chest, I need cleavage and for them to cover my chest like yours.

I have not had a recent blood test, my last one was still good, T is suppressed, Estradiol is over 200 pg/ml at trough so my dose was raised.

Have not had my DHT tested, I am on bicalutamide which is supposed to block DHT. I have also cycled red reishi because it's poison if you don't cycle it.

I have been trying every NBE method on this site, I have read all your posts.

I just think god made some people men and we aren't meant to look female and grow breasts, while people like you are made to grow breasts and any method will make your chest grow.

(28-11-2021, 10:29 AM)HandofFate Wrote: [ -> ]I just think god made some people men and we aren't meant to look female and grow breasts, while people like you are made to grow breasts and any method will make your chest grow.

I agree in part with the former, but the latter is a garbage statement...and a copout.

You should do more research before claiming reishi is poisonous. Reishi mushroom extract is Hepatoprotective...meaning it protects the liver. Reishi protects the liver mainly from its polysaccharides, antioxidants and free radical-scavenging activity. Here's only two studies (of many) on hepatoprotective activity of reishi.

Hepatoprotective Activity and the Mechanisms of Action of Ganoderma lucidum (Curt.:Fr.) P. Karst. (Ling Zhi, Reishi Mushroom) (Aphyllophoromycetideae)

Yihuai Gao, Min Huang, Zhi-Bin Lin, Shufeng Zhou

International Journal of Medicinal Mushrooms 5 (2), 2003

Herbal medicines are always considered to be a safe and useful approach for the treatment of chronic hepatopathy. Ganoderma lucidum (Curt.: Fr.) P. Karst.[(Ling Zhi, Reishi mushroom)(Aphyllophoromycetideae)], a highly ranked medicinal mushroom in Oriental traditional medicine, has been widely used for the treatment of chronic hepatopathy of various etiologies. Data from in vitro and animal studies indicate that G. lucidum extracts (mainly polysaccharides or triterpenoids) exhibit protective activities against liver injury induced by toxic chemicals (eg, CCl 4) and Bacillus Calmette-Guerin (BCG) plus lipopolysaccharide (LPS). G. lucidum also showed anti–hepatitis B virus (HBV) activity in a duckling study. Recently, a randomized placebo-controlled clinical study showed that treatment with G. lucidum polysaccharides for 12 weeks reduced hepatitis B e antigen (HBeAg) and HBV DNA in 25%(13/52) patients with HBV infection. The mechanisms of the hepatoprotective effects of G. lucidum have been largely undefined. However, accumulating evidence suggests several possible mechanisms. These include antioxidant and radical-scavenging activity, modulation of hepatic Phase I and II enzymes, inhibition of b-glucuronidase, antifibrotic and antiviral activity, modulation of nitric oxide production, maintenance of hepatocellular calcium homeostasis, and immunomodulating effects. G. lucidum could represent a promising approach for the management of various chronic hepatopathies. Further studies are needed to explore the kinetics and mechanisms of action of G. lucidum constituents with hepatoprotective activities.
https://www.researchgate.net/publication...eae_Review

Hepatoprotective Effects of Mushrooms

6.2. Triterpenoids, Polysaccharides and Peptides from Ganoderma lucidum

The most studied mushroom with respect to hepatoprotective effects is Ganoderma lucidum. This fact is not surprising because G. lucidum is, indubitably, the most studied medicinal mushroom. Approximately 400 chemical substances have been isolated from G. lucidum, which include mainly polysaccharides, triterpenoids, nucleosides, ergosterols, fatty acids, proteins/peptides, and trace elements. Particularly polysaccharide and triterpenoid components in G. lucidum have been proposed as the bioactive constituents responsible for the protective activities against toxin-induced liver injury [105,106,107]. In a broad review about the hepatoprotective properties of G. lucidum, Gao et al. [106] collected evidence to suggest possible molecular mechanisms to explain its hepatoprotective actions. Among these mechanisms, the authors include antioxidant and radical-scavenging activity, modulation of hepatic Phase I and II enzymes, inhibition of β-glucuronidase, antifibrotic and antiviral activity, modulation of nitric oxide production, maintenance of hepatocellular calcium homeostasis, and immunomodulatory effects.

Data from in vitro and animal studies indicate that G. lucidum extracts, mainly polysaccharides or triterpenoids, exhibit protective activities against liver injury induced by toxic chemicals (e.g., CCl4) and Bacillus Calmette-Guerin (BCG) plus lipopolysaccharide (LPS). The fungus also showed antihepatitis B-virus (HBV) activity in a duckling study. Recently, a randomized placebo-controlled clinical study showed that treatment with G. lucidum polysaccharides for 12 weeks reduced hepatitis B e antigen (HBeAg) and HBV DNA by 25% (13/52) in patients with HBV infection. The mechanisms of the hepatoprotective effects are still undefined. Evidence suggests that antioxidant and radical scavenging activity, modulation of hepatic phase I and II enzymes, inhibition of b-glucuronidase, antifibrotic and antiviral activity, modulation of nitric oxide production, maintenance of hepatocellular calcium homeostasis, and immunomodulatory effects might be involved [107].

The effects of total triterpenoids extracts from G. lucidum on two different experimental liver injury models induced by carbon tetrachloride and d-galactosamine were extensively studied in mice [35,40,108,109]. Administration of the extract (80 mg/kg) significantly inhibited the increase of serum ALT and liver triglyceride levels in the models, effects similar to those of malotilate, a known reference substance for this kind of protective effects [110]. The G. lucidum extract also antagonized the decrease of the SOD activity and the GSH content and inhibited the increase of the MDA content in the carbon tetrachloride and d-galactosamine liver-injured mice. It could equally improve the histopathological changes. These observations are likely to indicate that triterpenoids isolated from G. lucidum have a powerful protective effect against liver damage induced by carbon tetrachloride and d-galactosamine. Their hepatoprotective effects were perhaps related to the ability to increase the activity of free radical scavenging enzymes and, thus, to raise the ability of antioxidation. It should be stressed that ganoderic acid (Figure 2), one of the triterpenoids found in G. lucidum, was proven to be a potent inhibitor of β-glucuronidase activity, an indicator of hepatic damage [111].

The hepatoprotective activity of peptides from Ganoderma lucidum was evaluated against d-galactosamine (d-GalN)-induced hepatic injury in mice. G. lucidum peptides were administered via gavage daily for two weeks at doses of 60, 120 and 180 mg/kg, respectively. The d-GalN-induced hepatic damage was manifested by a significant increase in the activities of marker enzymes (AST, ALT) in serum, by the increased MDA levels in the liver, and by significant decreases in the activity of SOD and in the GSH level in the liver. Pretreatment of mice with G. lucidum peptides maintained these parameters at their normal values. These biochemical results were supplemented by histo-pathological examination of liver sections. The best hepatoprotective effects of the G. lucidum peptides were observed after treatment with the dose of 180 mg/kg as deduced from the biochemical parameters and liver histopathological examinations. Results of this study revealed that the G. lucidum peptides can produce a significant diminution of the d-GalN-induced hepatocellular injury [40].

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6270077/#!po=30.5310

If you don't have SHBG tested you won't be able to determine how much free E2 (or its percentage) is available to bind in tissues. You can actually have normal range E2 but low free E2, thus a very small percentage of free E2 is available to make a significant impact on breast growth or feminization.

Total estradiol (or even total T) only paints a partial picture...ask your doctor for this test. There's no reason or excuse why you shouldn't ask. I believe there's nothing else to discuss here until you have test results to share.

Breast Growth For Genetic Males

Phanatic

Lotus

HandofFate

HandofFate

Drew

Nipply Russel

HandofFate

Lotus

HandofFate

Lotus