Breast Growth For Genetic Males

Lol, perhaps I'm just a boob mechanic

(30-01-2015, 09:26 PM)Lotus Wrote: [ -> ]Lol, perhaps I'm just a boob mechanic

Yes sirrriii , I will roll with that
Lotus if you need any business planning / set up / loan project report / profitability plaNning you know where to come

(30-01-2015, 09:30 PM)myboobs Wrote: [ -> ]

(30-01-2015, 09:26 PM)Lotus Wrote: [ -> ]Lol, perhaps I'm just a boob mechanic

Yes sirrriii , I will roll with that
Lotus if you need any business planning / set up / loan project report / profitability plaNning you know where to come

Ok cool, and thanks, (I'll defo be talking to my new financial planner, you!) I do need a good tax preparation, Uncle Sam wants mo money.

(30-01-2015, 09:26 PM)Lotus Wrote: [ -> ]Lol, perhaps I'm just a boob mechanic

You should SOOO Change your user title to: Boob Mechanic

That would be awesome to see considering how much great information you post that is so helpful for everyone.

~Elain

Some important other facts about GT, I'm just in amazement of this study tbh, I mean who knew green tea had this much potential. Imo it was a minor supplement, and it had no real NBE value.

EGCG-activated eNOS pathways that improve cardiovascular function and anti-cancer effect in the presence of PDE5 inhibitor

Intracellular signaling pathways
In cell culture, the majority of [3H]–EGCG is found in the cytosolic fraction [34]. This suggests that biological actions of EGCG may occur through EGCG metabolites or interaction with intracellular molecules. As mentioned above, EGCG produces low level reactive oxygen species, including hydrogen peroxide that may act as a second messenger for downstream signaling pathways [23], [36], [64], [97] and [121]. This action may be mediated by direct chemical reactions of EGCG with compounds at the cell surface. However, additional unknown receptor-mediated signaling pathways cannot be excluded. EGCG also increases other intracellular second messengers including Ca2+, cAMP, and cGMP.

DNA methylation
EGCG has epigenetic functions in chromosomes [25]. Aberrant methylation on CpG islands cause gene silencing that leads to altered cellular physiology and cell proliferation. EGCG inhibits DNA methyltransferase (DNMT) which reverses methylation-induced gene silencing by directly binding to DNMT with an IC50 of less than 1 μM EGCG [26]. This suggests that EGCG is transported to the nucleus. Although this function of EGCG has been known for a decade, the specific genes affected by this mechanism are not well defined and this area requires further investigation.

Recent molecular, cellular, and animal studies have begun to reveal detailed mechanisms linking drinking green tea and life-style adjustment with prevention of chronic diseases including cancer, diabetes, and cardiovascular disorders. Furthermore, chemical modification of an EGCG pharmacophore may modify relative therapeutic activities so that combinatorial supplementation may synergistically enhance beneficial health effects [8] and [30].

(30-01-2015, 09:40 PM)Lotus Wrote: [ -> ]

(30-01-2015, 09:30 PM)myboobs Wrote: [ -> ]

(30-01-2015, 09:26 PM)Lotus Wrote: [ -> ]Lol, perhaps I'm just a boob mechanic

Yes sirrriii , I will roll with that
Lotus if you need any business planning / set up / loan project report / profitability plaNning you know where to come

Ok cool, thanks. I do need a good tax preparation, Uncle Sam wants mo money.

Oh but Tax preparation is easy. Open your Bank account and let the IRS take it all. Done. *nods wisely and points to empty bank account* They are always happy to take your money, not so willing to give it back. lol

so true Elain, isn't it a slogan on some bureaucrat's wall about always keeping the little guy down. oh, thanks for the suggestion, I needed a change, frankly I was bored with the old handle. oh I changed my mind

Anyways, I'll stop for now where I started originally, only I left part of it out in the beginning post, there's still much more, that's enough for now.

Hey my BN friends, there's a ton info to which I'm about to share, my goal is to keep it reasonably user friendly (I'll do my best not to loose yah).

We all have an idea what makes breast growth possible, so no need go into detail there. What optimizes breast growth can be somewhat of a mystery, I think I have some solutions, or strategies that can help. You'll see how inflammation, oxidative stress, circulation, fat metabolism/diet, bioavailability of hormones will be key in the areas of improvement for increasing breast growth. At the bottom is green tea, however I'd like to start with this underrated phenomenon of NBE.

1) relieving oxidative stress/inflammation
2) knowing your metabolism/oxidative type
3) building/transforming adipose (fat metabolism)
4) increasing blood oxidation (oxygenating the blood)
5) improving the bioavailability of hormones
7) the oil rub technique
8) free radicals
9) antioxidants
10) green tea

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1) relieving oxidative stress/inflammation
Oxidative stress, inflammation, and cancer: how are they linked?
http://www.ncbi.nlm.nih.gov/pubmed/20840865/

2) knowing your metabolism/oxidative type

What Is Oxidative Metabolism?
http://www.wisegeekhealth.com/what-is-ox...bolism.htm

The blood sugar diet
http://www.totalweightlosscenter.com/for...r_Diet.pdf

Balanced Oxidizer Diet
http://www.livestrong.com/article/315489...izer-diet/

http://www.drkaslow.com/html/fast_oxidizer_diet.html
3) building/transforming adipose (fat metabolism)

Introduction to Adipose Tissue (stem cell research)

http://themedicalbiochemistrypage.org/ad...tissue.php

4) increasing blood oxidation (oxygenating the blood)
Foods to Increase the Oxygen in the Blood
http://www.livestrong.com/article/344130...the-blood/

5) improving the bioavailability of hormones
Bioavailability: How the Nutrients in Food Become Available
http://www.healthguidance.org/entry/6265...odies.html

4 Serum bioavailability of sex steroid hormones
http://www.sciencedirect.com/science/art...5X8680024X
Serum bioavailability of sex steroid hormones.
http://www.ncbi.nlm.nih.gov/pubmed/3521955

6) the oil rub technique

See entry post 1181 & 1182

7) free radicals

Free-radical theory of aging
http://wikipedia.org/wiki/Free-radical_theory_of_aging

8) antioxidants

http://wikipedia.org/wiki/Antioxidant

(30-01-2015, 09:40 PM)Lotus Wrote: [ -> ]

(30-01-2015, 09:30 PM)myboobs Wrote: [ -> ]

(30-01-2015, 09:26 PM)Lotus Wrote: [ -> ]Lol, perhaps I'm just a boob mechanic

Yes sirrriii , I will roll with that
Lotus if you need any business planning / set up / loan project report / profitability plaNning you know where to come

Ok cool, and thanks, (I'll defo be talking to my new financial planner, you!) I do need a good tax preparation, Uncle Sam wants mo money.

Tax preparation !
Don't even go there I got accountant to do all my books / wages / tax

I couldn't resist with all this tax talk

[attachment=8811]

Thanks Froger, I needed a good laugh.

I intended to add more of the new info today but we are experiencing some technical issues (its being addressed).

However I will attempt to slip this new pathway in while I can. And it's called "pentose phosphate pathway" or "PPP" (btw my pervy friends that's not a penthouse thing).

PPP occurs exclusively in the cytoplasm, and is found to be most active in the liver, mammary gland and adrenal cortex in the humans.

The primary results of the pathway are:

The generation of reducing equivalents, in the form of NADPH, used in reductive biosynthesis reactions within cells (e.g. fatty acid synthesis).

Production of ribose-5-phosphate (R5P), used in the synthesis of nucleotides and nucleic acids.

Production of erythrose-4-phosphate (E4P), used in the synthesis of aromatic amino acids.

Aromatic amino acids, in turn, are precursors for many biosynthetic pathways

Dietary pentose sugars derived from the digestion of nucleic acids may be metabolized through the pentose phosphate pathway, and the carbon skeletons of dietary carbohydrates may be converted into glycolytic/gluconeogenic intermediates.

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The potential benefit.

The following article is circulated throughout the body building world. Aside from that, you probably won't find anybody drawing the conclusion the a potential advancement of technologies exists here in the nbe world lol, except maybe yours truly.

The point on this is we can say that after PE workouts when the body is in recovery mode estrogen plays a significant role in recovery. Now imo, utilizing this new PPP pathway, adding fatty acids/polyphenols (green tea) afterwards will help the growth phase of breast growth. If you remember reducing inflammation and oxidative stress is an excellent way to also produce favorable breast growth potential.

Glucose Utilization and Estrogen
Estrogen may play an even more vital role in promoting an anabolic state by affecting glucose utilization in muscle tissue. This occurs via an altering the level of available glucose 6-phosphate dehydrogenase. G6PD is an important enzyme in the support anabolism, as it is directly tied to the use of glucose for muscle growth and recuperation[viii] [ix]. During the period of regeneration after skeletal muscle damage, levels of G6PD are shown to rise dramatically. G6PD enzyme plays a vital role in what is known as the pentose phosphate pathway, and as such this rise is believed to enhance the PPP related process in which nucleic acids and lipids are synthesized in cells; fostering the repair of muscle tissue.

A 1980 study at the University of Maryland has shown that levels of glucose 6-phosphate dehydrogenase rise after administration of testosterone propionate, and further that the aromatization of testosterone to estradiol is directly responsible for this increase.[x] In this study neither dihydrotestosterone nor fluoxymesterone could mimic the affect of testosterone propionate on levels of G6PD, an affect that was also blocked by the addition of the potent anti-aromatase 4-hydroxyandrostenedione to testosterone. 17-beta estradiol administration caused a similar increase in G6PD, which was not noticed when its inactive estrogen isomer 17-alpha estradiol (unable to bind the estrogen receptor) was given. An anti-androgen could also not block the positive action of testosterone. This study provides one of the first palatable explanations for a direct and positive effect of estrogen on muscle tissue.

What does this all mean?
It is a long held belief among athletes that estrogen maintenance drugs can slightly hinder muscle gains during steroid therapy with a strong aromatizable steroid such as testosterone. Whether or not we have plausibly explained this remains to be seen, however the above evidence certainly does provide strong support for a direct and positive affect of estrogen on growth. Does this mean we should abandon estrogen maintenance drugs? I don’t think that should be the case. It is important to remember that estrogen can deliver many unwanted effects such as increased water retention, fat deposition and the development of female breast tissue when it becomes too active in the male body. Clearly if we plan a high-dose cycle with an aromatizable steroid, anti-estrogens will be an important inclusion. However we cannot ignore the suggestion of using estrogen maintenance drugs only when they are necessary to combat visible side effects during mild to moderately dosed cycles,especially if bulk is the ultimate goal of the athlete.

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Glucose-6-phosphate dehydrogenase (G6PD or G6PDH)

There is an enzyme present in the pentose phosphate pathway , Glucose-6-phosphate dehydrogenase (G6PD or G6PDH) a metabolic pathway (chemical reaction) that supplies reducing energy to cells (such as erythrocytes) by maintaining the level of the co-enzyme nicotinamide adenine dinucleotide phosphate (NADPH). The NADPH in turn maintains the level of glutathione in these cells that helps protect the red blood cells against oxidative damage. Of greater quantitative importance is the production of NADPH for tissues actively engaged in biosynthesis of fatty acids and/or isoprenoids, such as the liver, mammary glands, adipose tissue, and the adrenal glands. G6PD reduces nicotinamide adenine dinucleotide phosphate (NADP) to NADPH while oxidizing glucose-6-phosphate.