[Dianna1395]

Hi spanky, happyme and crew.

Reduced sperm (quite possibly permanently). Dustasteride inhibits two types of 5 alpha reductase (type I & II), it'll tank total and free T. It looks like it has a surge (of T) in the first 30-45 days, most likely from negative feedback of lutenizing hormone. This surge (or flare as they say) also happens to a lesser extent when starting PM, and from my experience BO did the same thing at first. Lowered T can have a strong impact on libido, lowered free T will impact penis size.
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Lotus,
That makes the mad scientist in me wonder, could we "manage" free T, increase E, and in essence make the best of both worlds?
I somewhat doubt it, but, would be interesting to look into it.
Eventually, though, the E kills the T (Negative feedback on APT loop, I think it is; suppresses Leutenizing hormone, per female cycle.)

But with our cyclic approaches anyway, might be able to accomplish something. :-)

Your thoughts?
-Dianna

Hey there demonid P, what up B?. (j/k) Lmao

Ok I dig where you're going, and I've spent amount of time thinking about this lol, meaning the idea of boosting free T to benefit biosynthesis of E. Btw, Pharma E2 inhibits DHT (I don't know the exact amount as of yet, (I will though lol).

Negative feedback is a good example, in initial treatment phase some meds increase lutenizing hormone before it (med) stabilizes (30-45 days). To me, this says the science is there to manipulate meds.

Something I'm close to understanding is using our diurnal rhythms to promote growth stages that can boost feminization (similar to some animal based studies). And I think amino acids & peptides in combination with a few other things I'll mention later will trigger this boost.

Hi, Lotus,
I'm probably going in a different direction from you on the Free T line of thought.
I think it would be great to have the Free T for playing with the woman (a woman), and the breasts grow in from aromatization of the Free T. So, control the DHT by kill 5-Alpha-R, which would likely mean Dutas... (But I need to review if that kills T production, or just wipes out the 5Ar). Then using our other goodies, like GTE, Spearmint, Red Clover, PM, WP, induce an environment high in aromatase, high in E, but still forcing E into the system. (How to induce high LH is a question; thinking I'd have to use Clomiphene citrate to rev up the leydig cells... Or, find a way to stimulate ONLY LH, as FSH will cause other, unwanted effects.

It's indicated here though (not a scientific site, though: http://androcycle.com/post-cycle-therapy/ - I don't have much capacity to do research from behind the NWO corporate firewalls, category "illegal drugs" is not allowed. Effing tyrants, both corp and DEA/FedGov.)

Or maybe, from http://www.mayoclinic.org/diseases-condi...n-20033618 - there are a few "shortcuts" to my preferred end goal? Ah, if only... (there were this site in 1990, the internet had spread to Thailand, the gov't wasn't busy coralling us like cattle, and my parents weren't self-righteous abusive pricks...) ;-)

OTOH... Maybe using something else to block FSH, or to eliminate it, would work? And as an add-on, what about finding a means to ensure Prostate stays "normal" and thus allow for a female experience even with the estrogen...? Never thought about that before, but since Prostate is tied to DHT, prob'ly not. But maybe there's something like Clometipene (?) which would not block the creation of the T, but rather interfere with the connectors, so the T cannot be used.... But then that would affect everywhere else, too. So, no erections while on the meds. :-P

Easier to just get an inflatable phallus! :-)

-Dianna