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(26-12-2015, 11:21 AM)WendyA Wrote:  With this sense of humor I take it coronary artery disease wasn't the cause of your heart surgery.

All I can say is that I know as much as the doctor's do. July of the same year I had my heart attack, I did a tread mill stress test. The doctor said since I just turned 50, it would be nice for that as a routine.

The goal, was to stress my heart enough to get to like a 120 BPM. After being on the tread for 20 mins, they introduced a drug to put more stress on me. They were amazed that I couldn't break 100BPM after 20 min's at 2 MPH. (been an decent athlete most of my life)

The Cardiologists said I had a heart of a 25 yr old. 6 months later that same doctor was there when I arrived in an ambulance. So, you tell me. I am sure your thoughts, idea's and prejudices is as good as anyone I have ran into...TongueBig Grin
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(26-12-2015, 03:25 PM)iaboy Wrote:  All I can say is that I know as much as the doctor's do. July of the same year I had my heart attack, I did a tread mill stress test. The doctor said since I just turned 50, it would be nice for that as a routine.

The goal, was to stress my heart enough to get to like a 120 BPM. After being on the tread for 20 mins, they introduced a drug to put more stress on me. They were amazed that I couldn't break 100BPM after 20 min's at 2 MPH. (been an decent athlete most of my life)

The Cardiologists said I had a heart of a 25 yr old. 6 months later that same doctor was there when I arrived in an ambulance. So, you tell me. I am sure your thoughts, idea's and prejudices is as good as anyone I have ran into...TongueBig Grin

You describe the often typical presentation of coronary artery disease. Your heart can be functioning fine then a heart attack. Hence the saying that you can't outrun a bad diet. For about 40% of folks their first symptom of coronary artery disease is the heart attack that kills them.

If I were in your situation I would be following Dr. Caldwell B. Esselstyn Jr. guidance on how to live a healthy life post heart attack with no more symptoms. I currently choose to follow his advice to prevent this disease to begin with. His multi decade case study conducted at The Wellness Institute of the Cleveland Clinic has shown less then a 1% recurrence of any symptoms (post heart attack) from those following his advice and no coronary artery disease deaths.

With-in the first 15 minutes of this video he outlines exactly what you may have experienced.


That said I'm not a doctor, I just don't want to experience coronary artery disease the same as my Dad. Following Dr. Esselstyn's advice gives me total control over this disease.

Jaimela Dulaney is a cardiologist who uses plant based nutrition to treat her patients. She has a podcast where she discusses much of her experience and interviews folks from all walks of life. You might find it informative.

I wish you well, any further discussion will have to be in a new thread or through PM. Peace!
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(26-12-2015, 03:25 PM)iaboy Wrote:  
(26-12-2015, 11:21 AM)WendyA Wrote:  With this sense of humor I take it coronary artery disease wasn't the cause of your heart surgery.

All I can say is that I know as much as the doctor's do. July of the same year I had my heart attack, I did a tread mill stress test. The doctor said since I just turned 50, it would be nice for that as a routine.

The goal, was to stress my heart enough to get to like a 120 BPM. After being on the tread for 20 mins, they introduced a drug to put more stress on me. They were amazed that I couldn't break 100BPM after 20 min's at 2 MPH. (been an decent athlete most of my life)

The Cardiologists said I had a heart of a 25 yr old. 6 months later that same doctor was there when I arrived in an ambulance. So, you tell me. I am sure your thoughts, idea's and prejudices is as good as anyone I have ran into...TongueBig Grin
Runs off frantically searching for her coenzyme q10!
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Sorry Lotus, didn't think one smart alec fun reply about steaks would derail your program thread. Huh
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(24-12-2015, 12:08 PM)hannah Wrote:  Great to see you're active again, you keep things alive in here with your interesting posts, I wouldnt wanna miss a Lotus thing:p
Anyhow just one question as you're here now, my dogs get sheep-fat with dried aglues from me and I snack from it too for the fat..I think it might be good for NBE,but what do you think about eating bad fats> what about the amounts?
I alreaady take so much,breads with olive oil, 2 spoons C.O...where does the fat in fat out stops? Or are fats healthy,there's a lot of controversial info about it..

Hi Hanah, (thanks hun) Wink

Good fats vs bad fats?, good question.....bad fats raise LDL (think processed foods), trans fats, saturated fats (although coconut oil is actually beneficial). Some fats (polyunsaturated-omega 6's) inhibit DHT, EPO, walnuts (my NBE #2, coconut oil is #1) come to mind. Omega 3 (fish oil) raises free testosterone in men and helps improve central obesity in women, I also believe it helps fight estrogen dominance.

Without fat (new fat) I think breast growth is less likely to progress. Fat in' refers to new fat, in other words, using this new energy to burn existing fat towards NBE goals, "Fat out". I like to think of it as 'Fat Cell Technology', or manipulating fat cells to synthesize with hormone steroids for targeted cell growth. Big Grin
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Hi BN,

A quick thanks to Marcy, Ella, Hannah, Pom , Eloise (omg thanks Big GrinTongue), Wendy, iaboy (no worries, post away), thank you again.........Cool

I keep going back to Leptin and how it can help benefit NBE. Additionally, "adipose" (fat) is also interwoven with a huge endocrine function, number #3 in my book. My apologies for the technical nature of this post, please plod along the best you can, Big Grin if you can grasp some of the basics of this (LEPTIN and adipose) you will undoubtedly be further along on how to adopt it into NBE. I will provide some of the broad strokes, (hopefully). RolleyesTongue may the Boobie Force be with you. Wink

Aromatization of Androgens by Human Abdominal and Breast Fat Tissue
Abstract
The ability of human abdominal, breast and axillary fat to convert androgens into estrogens was investigated by incubating labeled substrates in the presence of NADPH with a variety of cell preparations. The incubation products were subjected to phenolic partition, paper chromatography, methyl-ether formation, repeat chromatography and crystallization with cold carrier reference standards to constant specific activity. Androstenedione was converted to estrone and, to a lesser extent, to 17β-estradiol by crude homogenates, minces, fat-free particulate fractions (1,000–100,000 × g) and isolated fat cells obtained from abdominal, breast or axillary fat. Testosterone was found to be aromatized as actively as androstenedione, but in this case more 17 β-estradiol was formed than estrone. 19-Hydroxyandrostenedione2 also served as substrate, giving results similar to those obtained with androstenedione. Fat tissue obtained from cancerous breasts was found to be as active as normal breast fat (1–4 pg/g fat/90 min) and within the range found for abdominal fat (1–27 pg/g fat/90 min). In each case in which axillary fat was compared to breast fat from the same subject, the activity of the axillary fat was 5 to 10 times higher. The results indicate a possible role of adipose tissue as a significant extra-gonadal source of estrogens.[/b]
- See more at: http://press.endocrine.org/doi/abs/10.12...Mhqtw.dpuf


From the study below:

Several steroidogenic enzymes are expressed in adipose tissue including cytochrome P450-dependent aro- matase, 3 -hydroxysteroid dehydrogenase (HSD), 3 HSD, 11 HSD1, 17 HSD, 7 -hydroxylase, 17 -hydroxylase, 5 - reductase, and UDP-glucuronosyltransferase 2B15 (71, 72). Given the mass of adipose tissue, the relative contribution of adipose tissue to whole body steroid metabolism is quite significant, with adipose tissue contributing up to 100% of circulating estrogen in postmenopausal women and 50% of circulating testosterone in premenopausal women.

Although the adrenal gland and gonads serve as the primary source of circulating steroid hormones, adipose tissue expresses a full arsenal of enzymes for activation, interconversion, and inactivation of steroid hormones.
http://www.iub.edu/~k662/articles/obesit...202004.pdf


Adipose Tissue as an Endocrine Organ

Adipose tissue is a complex, essential, and highly active metabolic and endocrine organ. Besides adipocytes, adipose tissue contains connective tissue matrix, nerve tissue, stromovascular cells, and immune cells. Together these components function as an integrated unit. Adipose tissue not only responds to afferent signals from traditional hormone systems and the central nervous system but also expresses and secretes factors with important endocrine functions. These factors include leptin, other cytokines, adiponectin, complement components, plasminogen activator inhibitor-1, proteins of the renin-angiotensin system, and resistin. Adipose tissue is also a major site for metabolism of sex steroids and glucocorticoids. The important endocrine function of adipose tissue is emphasized by the adverse metabolic consequences of both adipose tissue excess and deficiency. A better understanding of the endocrine function of adipose tissue will likely lead to more rational therapy for these increasingly prevalent disorders. This review presents an overview of the endocrine functions of adipose tissue. (J Clin Endocrinol Metab 89: 2548 –2556, 2004)
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Btw, it's exciting seeing all the new members (too many to list), Tongue I hope you all find the information you seek and build new friendships along the way......(and of course build new boobage too). Big Grin

Also, I see congratlations are in order to veterans on their continued growth, what do they say?, good things come to those who wait. Big Grin........awesome.
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Hi d d ..
So how do you do the whole leptin thing?
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(30-12-2015, 07:31 AM)ellacraig Wrote:  Hi d d ..
So how do you do the whole leptin thing?

Hi Ella, great question, I'm glad you asked.

Leptin (a fat burning hormone) burns fat and then makes the mitochondria full (or, a fed state) thereby increasing the chance of weight loss. And oh btw, mitochondria is the main energy in cells.

In other words, instead a new fat being stored, (making us fat) it is directly synthesized into usable energy, e.g. aromatase x20, usable "stored" energy from old fat into NEW released estrogen. It's sometimes referred to as "liberating fat".

Here's a closer look, but I have other ideas Tongue of how Leptin can work for NBE........DodgyRolleyes

Leptin Turns Fat Cells Into Fat-Burning Cells
http://www.webmd.com/diet/20040209/lepti...ning-cells
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A thinner person might have Less Leptin, making them feel sluggish, proper sleep 7-8 hours builds natural Leptin. Weight loss supplements claim to have Leptin, but that's not really the case. Increasing Leptin intake by certain foods?, some might influence, I would say fat burning supplements can also aid in increasing Leptin back into cells. Research Chem?, yeah possible but that's too risky imo.
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