Hey Lotus. Thanks for coming in with calm responses. For starters I have collected all of your messages on X thread and extracted structured statements and links that I use with chatgpt o1 pro to analyze them and get additional reasoning and data. Don't be distracted by my low post count.
I have a few questions;
1. I have a feeling that your cream is not 10% of E2, but 10% of 0.06% gel. Because otherwise 1g of cream would be 100mg of topical E2 that would yield insane local blood levels. Am I right?
2. DHEA efficiency looks to be very dependent on genetics and in my case it produces tons of local androgen. Do you agree that in such case it should be avoided?
3. Am I right that you believe that RC is the most effective PPAR-Y agonist? And if so, how do we know it's not competing for receptors with e2?
Thank you
I have a few questions;
1. I have a feeling that your cream is not 10% of E2, but 10% of 0.06% gel. Because otherwise 1g of cream would be 100mg of topical E2 that would yield insane local blood levels. Am I right?
2. DHEA efficiency looks to be very dependent on genetics and in my case it produces tons of local androgen. Do you agree that in such case it should be avoided?
3. Am I right that you believe that RC is the most effective PPAR-Y agonist? And if so, how do we know it's not competing for receptors with e2?
Thank you

