[Lotus]

Hi BN, after many hours, days and years of research on BO I've found some key pieces of information on how using BO could have better results growing breasts. My approach to finding this information looked at the bovine ovary and granulosa cells. So, bovine ovary promotes aromatase (via the CYP19 enzyme & cAMP dependent signaling pathway. It also stimulates IGF-1, and probably the biggest discovery on BO is how it stimulates Sirtuins, and its family (see study below). Sirtuins are powerful, it's been reported how they extend lifespans. 

So how I'd use BO is with the topicals, meaning with DHEA, E2 (cream or gel) and Progesterone cream. I'd also recommend using a Nipple Pump to facilitate lactation. I've tried all of the above and can say it works. I've used some of the best electric breast pumps out there, and the one I'll share tomorrow works the best for me. I will say I've converted breast pumps to nipple pumps and get some of the best overall lactation results. 

There's other information to share about BO but I've run out of time (or gas). 

Granulosa cells (GC) are critical regulators of ovarian function and fertility. 

Effects of Follicle-Stimulating Hormone, Insulin-Like Growth Factor 1, Fibroblast Growth 
Factor 2, and Fibroblast Growth Factor 9 on Sirtuins Expression and Activity in Bovine Granulosa Cells

Abstract
Granulosa cells (GC) are critical regulators of fertility. During the process of ovarian folliculogenesis, these cells undergo profound changes while producing steroid hormones that are important to control follicular growth, oocyte maturation, and ovulation. Sirtuins are enzymes that regulate several biological processes and have been associated with control of GC function. However, how sirtuins are regulated in GC during ovarian folliculogenesis remains to be unveiled. The present study was designed to investigate effects of hormones that control GC proliferation, differentiation, and steroidogenesis on expression of the seven members of the mammalian sirtuins family (SIRT1-7) and on histone deacetylase activity of nuclear sirtuins (SIRT1, 6, and 7) in GC. Bovine granulosa cells were isolated from small antral follicles (1-5 mm) and were treated with or without follicle-stimulating hormone (FSH), insulin-like growth factor 1 (IGF-1), and fibroblast growth factors 2 (FGF2) and 9 (FGF9). Following treatments, cell proliferation was determined via a cell analyzer, estradiol synthesis and histone deacetylase activity were determined via ELISA, and sirtuins mRNA expression was determined via qPCR. Treatments with FSH and IGF-1 stimulated cell proliferation while addition of FGF2 or FGF9 suppressed estradiol production stimulated by FSH plus IGF-1. In terms of treatments that regulated sirtuins expression in GC, fibroblast growth factors were the most impactful: FGF2 alone increased SIRT1 mRNA expression in comparison to several treatments and increased mRNA abundance of SIRT2 and SIRT7 when added to the combination of FSH and IGF-1; the addition of FGF9 to the combination of FSH and IGF-1 increased mRNA expression of SIRT2, SIRT3, SIRT4, SIRT6, and SIRT7 and increased mRNA expression of SIRT5 in comparison to the negative control group that received no treatment. Also, FGF2 alone increased histone deacetylase activity of sirtuins in comparison to all treatments that contained FSH and/or IGF-1. Furthermore, several correlations were observed between treatments and sirtuins expression and activity, between estradiol or GC numbers and sirtuins expression, and between expression of sirtuins. As FGF2 and FGF9 are considered anti-differentiation factors of GC that stimulate GC proliferation while suppressing estradiol production in combination with FSH and IGF-1, data of this study suggest that sirtuins are associated with control of differentiation of bovine GC.

High Levels of SIRT1 Expression as a Protective Mechanism Against Disease-Related Conditions
In literature, it was shown that sirtuins' hyperactivity could reduce these negative outcomes both in vivo and in vitro due to its neuroprotective role (68–71)
https://www.frontiersin.org/journals/end...00614/full

The 5′-Flanking Region of the Ovarian Promoter of the Bovine CYP19 Gene Contains a Deletion in a Cyclic Adenosine 3′,5′-Monophosphate-Like Responsive Sequence
https://academic.oup.com/endo/article/13...ogin=false

Effects of IL-11/IL-11 Receptor Alpha on Proliferation and Steroidogenesis in Ovarian Granulosa Cells of Dairy Cows
Taken together, these data indicate that the effects of IL-11/IL-11Rα on the proliferation and steroidogenesis in bovine GCs is mediated by the JAK1-STAT3, PKA-CREB, p38 MAPK, and ERK1/2 signaling pathways. Our findings provide important insights into the local action of the IL-11 system in regulating ovarian function.

Weezer, thank you for your kind words. As for the skin healing with DHEA, you can find additional research at the bottom of each research link. 

Hi Lara, yes… 50 mg of DHEA taken orally produces more aromatase, but not to DHEA's metabolites: 
androst-5-ene-3beta,17beta-diol (ADIOL), androst-5-ene-3,17-dione, and androst-4-ene-3,17-dione were activators of PXR and (PPAR) subfamily.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2423429/

Sweets, apologies, I'll share (hopefully tomorrow) why more tests are being run.