[Lotus]

Thanks for this info Lotus. So as you said we don't want to take more calorie in, since it will go to wrong places, then how do we direct it toward a certain area? Thanks, POM

Fats get broken down through the intestines and into the lymphatic/blood system. And oh btw, massaging breasts breakdowns fats into the lymphatic system and excreted. Which tells me massaging before you pump should be of more benefit, meaning, you don't want the fats you just stretched to be broken down and excreted, right?..........that's just my opinion. as I try to explain this, and no doubt it will be complex, I'd like to point out that the hormone Leptin (once again is a major player in breast growth, (think receptors, which Leptin receptors are in the breasts). Additionally, LEPTIN is present in breast milk, do we see a pattern here?. I will remind you that Aromatase is stimulated in the breasts by?????.....what else, LEPTIN.

Anyways, fats are broken into fatty acids which stimulate tissues, (taken from the reseach) e.g. when fats are being transported in the lymph or blood, large fat molecules need to be broken down into smaller fatty acids. The larger fats do not "attract" as many excess water molecules by osmosis as many smaller molecules would. Under the stimulation of insulin, these fatty acids are made into fat molecules and stored as fat droplets, aka, into tissues). And this is where it gets interesting......

Researchers identify ‘fat gene’ associated with obesity - See more at: http://news.uchicago.edu/article/2014/03...sity#.dpuf

Researchers isolate a gene that controls body mass and regulates body composition, which has an ability to be resistant to a high Fat induced diet, in other words it handles glucose better and protects against diabetes C.

Hypothalamic function of IRX3, therefore, appears to control body mass and composition in these animals, indicating that the genetic predisposition to obesity is wired in the brain.

FTO gene-Fat mass and obesity-associated protein also known as alpha-ketoglutarate-dependent dioxygenase FTO is an enzyme that in humans is encoded by the FTO gene located on chromosome 16.
https://en.m.wikipedia.org/wiki/FTO_gene

exercise more, eat more= (partial growth)
restrict calories = slow metabolism (no growth)
add intermittent fasting mixed with HIIT=growth hormone
energy from fat stores is highly concentrated

This is where Ketosis (or ketogenesis) comes in, nearing starvation the body goes into a ketogenic state. Ketogenesis doesn’t destroy muscle tissue, but is rather the process by which stored fat is turned into ketones — a perfectly usable energy source for every major body system.

The body fat setpoint is the mass of body fat that your body attempts to defend against changes in either direction. It’s your body’s attempt to maintain homeostasis.

HOW TO BURN STORED BODY FAT — A KETOSIS PRIMER
http://www.foodrenegade.com/how-to-burn-...is-primer/

Ok, I understand. I'll put it this way, you've had the benefit of some glandular growth (with awesome puffies) .....You want fat distribution to the breasts, correct?. Ok then, getting fat to peripheral tissues (breasts) without causing obesity is the desired outcome? (I'm guessing).

This (breast growth) is using a (or tuning) a multiple of hormones/proteins/fats/exercise/fasting. Simply put, I think we need to be in constant state of Flux (otherwise known as FAT FLUX) state where we add and burn fat while modulating hormones and proteins. In other words Breast growth comes from a stimulation of hormones (other things I noted, nerd stuff) and an increase of fat cells, (longer, elongated and rounded in that makes sense). This is simialr state of ketosis, but a modified state of ketosis (imo) it's something talked about but not fully understood, I've been thinking (trying to figure it out) about this for sometime.

We want healthy breast growth without the complications of weight, DVT, stress on our livers, cancer, etc. Liberatiing fat cells in peripheral tissues seems a likely prospect. Help is welcomed to add to this conversation.

FAT FLUX: enzymes, regulators, and pathophysiology of intracellular lipolysis
http://onlinelibrary.wiley.com/store/10.15252/emmm.201404846/asset/emmm201404846.pdf?v=1&t=iif6phme&s=4bb1f679be867a087e2bc66597ee5eb8d003becc

Progesterone 1/4 teaspoon daily, (applied to breasts [not nipples, which causes freckles on the areolas]).
Green tea 2-3 cups extract to promote Leptin (or green coffee bean extract)
Coconut oil (1-2 tablespoons) to help synthesis of new fat cells
Fat in, Fat out----that fat flux I described (e.g. activation of Lipogenesis of fat cells, then followed by NBE/ Hrt and then exercise, HITT preferably).

So sorry Bielz, I'm honestly not trying to confuse, I think it's the direction new breast growth needs to take. Btw, Leptin is the satiety hormone (tells we are full) and ghrelin is the hunger hormones (tells us to eat), if too much ghrelin is produced Leptin is inactivated, which means estrogen is not being activated to synthesize aromatase, in other words a missed opportunity of free growth.