Two ingredients that are vital in a NBe plan are green tea and coconut oil. coconut oil helps to bind estrogen receptors.
Some recent research show Aromatase expression in homogenates from scAT was 213% higher. In other words, aromatase in subcutaneous fat is 213% higher.
Br J Nutr. 2014 May 28;111(10):1782-90. doi: 10.1017/S000711451400004X. Epub 2014 Feb 11.
Influence of virgin coconut oil-enriched diet on the transcriptional regulation of fatty acid synthesis and oxidation in rats - a comparative study.
Arunima S1, Rajamohan T1.
Author information
Abstract
The present study was carried out to evaluate the effects of virgin coconut oil (VCO) compared with copra oil, olive oil and sunflower-seed oil on the synthesis and oxidation of fatty acids and the molecular regulation of fatty acid metabolism in normal rats. Male Sprague-Dawley rats were fed the test oils at 8 % for 45 d along with a synthetic diet.
Dietary supplementation of VCO decreased tissue lipid levels and reduced the activity of the enzymes involved in lipogenesis, namely acyl CoA carboxylase and fatty acid synthase (FAS) (P< 0·05). Moreover, VCO significantly (P< 0·05) reduced the de novo synthesis of fatty acids by down-regulating the mRNA expression of FAS and its transcription factor, sterol regulatory element-binding protein-1c, compared with the other oils. VCO significantly (P< 0·05) increased the mitochondrial and peroxisomal β-oxidation of fatty acids, which was evident from the increased activities of carnitine palmitoyl transferase I, acyl CoA oxidase and the enzymes involved in mitochondrial β-oxidation; this was accomplished by up-regulating the mRNA expression of PPARα and its target genes involved in fatty acid oxidation.
In conclusion, the present results confirmed that supplementation of VCO has beneficial effects on lipid parameters by reducing lipogenesis and enhancing the rate of fatty acid catabolism; this effect was mediated at least in part via PPARα-dependent pathways. Thus, dietary VCO reduces the risk for CHD by beneficially modulating the synthesis and degradation of fatty acids.
Suppression of Androgen Receptor Signaling and Prostate Specific Antigen Expression by (−)-Epigallocatechin-3-Gallate in Different Progression Stages of LNCaP Prostate Cancer Cells
http://www.ncbi.nlm.nih.gov/pmc/articles...s95453.pdf
The green tea polyphenol, (−)-epigallocatechin-3-gallate (EGCG), inhibits the development and progression of prostate cancer in TRAMP mice and in men. We examined the effects of EGCG on LNCaP human prostate cancer sublines 104-S, 104-R1 and R1Ad representing different progression stages of prostate cancer. EGCG suppressed cell proliferation, prostate specific antigen (PSA) expression, and AR transcriptional activity in the different LNCaP sublines. Intraperitoneal administration of EGCG also suppressed the growth of relapsing R1Ad tumors and decreased tumor- derived serum PSA. Effects of EGCG on tumor PSA expression have the potential to affect accurate monitoring of patient tumor burden by serum PSA measurements.
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50% decrease in serum PSA
EGCG inhibits cell proliferation, PSA and Human tissue kallikreins 2 (hK2) secretion, expression of AR mRNA, and transcriptional activity of AR in androgen-dependent LNCaP cells treated with androgen [18,19]. EGCG, but not EGC, ECG, or EC, suppresses tumor growth of androgen receptor (AR)-negative androgen-insensitive PC-3 prostate xenografts, AR-positive androgen-independent LNCaP 104-R1 prostate xenografts, and MCF breast xenografts in athymic mice [1].
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Plasma 17b-estradiol and testosterone concentrations. The plasma concentration of 17b-estradiol was undetectable in control rats, whereas that of GT-treated rats was 39.2 6 10.4 pmol/L (P , 0.01).
Aromatase expression. Relative aromatase expression in homogenates from scAT was ;213% higher than that of vAT (P , 0.05) in control rats (Fig. 2). Interestingly, GT treatment induced a marked increase in aromatase expression in both AT locations to 318.5 6 60.6% of control in scAT (P , 0.05) and 285.5 6 82.9% of control in vAT (P , 0.01).
We monitored ad libitum consumption of both food and fluid every other day and recorded rat weight every week. To prepare GT infusion, 1 L of boiling water (100°C) was dispensed over 3 tea bags (1.3 g/bag) for 5 min. The infusion was allowed to cool to room temperature before being transferred to the bottles and supplied to the rats.
has buckets of bras, but I love her collection. 
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